Is vitamin C good for diabetics? Vitamin C is a vitamin found in various foods and sold as a dietary supplement. It is used to prevent and treat scurvy. Vitamin C also plays an important role in collagen synthesis, wound healing, and iron absorption. While there is no conclusive evidence linking vitamin C with any disease state, some studies have shown that people who consume adequate amounts of vitamin C tend to be healthier than those who don’t.
For example, some research shows that higher serum concentrations of vitamin C may reduce risk of heart attack and stroke. However, not all trials on vitamin C have been positive. There is insufficient data to determine if vitamin C affects risk factors for coronary artery disease such as cholesterol levels, triglyceride. In this blog, we also have best vitamins and supplement for diabetics that you might want to see.
Is Vitamin C Good For Diabetics
Vitamin C deficiency is a common problem among diabetics. A higher sorbitol blood level, which can be detrimental and contribute to diabetic retinopathy and kidney damage, can be controlled by increasing one’s consumption of Vitamin C.
Additionally, vitamin C can enhance insulin sensitivity, lowering blood sugars, and improving HbA1c levels in persons with diabetes. In addition to fruits and vegetables, vitamin C may be found in supplements such as kiwi and bell peppers, as well as in guava, tomatillo, sweet potatoes, strawberries, spinach, and broccoli.
Having too much glucose in your blood, or blood sugar, can lead to diabetes. Blood glucose is your main source of energy and comes from the food you eat. Insulin helps control your blood glucose levels.
There are two types of diabetes: Type 1 Diabetes & Type 2 Diabetes. When insulin isn’t working properly, your body can’t use it properly or use other parts of the brain to get energy. This causes a buildup of glucose in your bloodstream.
Your body will do what it has learned through experience, it releases hormones or makes more insulin to try to lower this build-up of sugar in the blood. If these efforts aren’t enough, your pancreas may release less insulin over time. This condition is known as type 2 diabetes.
What Cause Diabetes?
Type 1 diabetes results when your immune system destroys cells that produce insulin. It usually starts when you’re young, although sometimes after age 40 it’s diagnosed later because symptoms don’t show up until then. In most cases, it’s not clear why an individual develops type 1 diabetes.
Some researchers think that certain viruses such as rubella virus, mumps virus, chicken pox virus and cytomegalovirus may trigger autoimmune destruction of beta cells. Researchers also suspect that genetics could be involved. There appears to be no relationship between birth order and risk of developing type 1.
However, the risk increases if either parent had the disease during their childhood. The likelihood of having type 1 diabetes is about one in 200 people worldwide and in 1 out of every 400 people living in North America.
Some researchers believe that some genes are associated with increased risk of type 1 diabetes. These include those on chromosome 6p, which have been linked to autoimmunity, especially in children; those on chromosomes 10q and 14q, which contain genes important for regulating T cell activity ; and those on chromosome 11p15,where a gene called HLA DQA1 has been found to increase the risk of type 1 diabetes.
Type 2 diabetes occurs when your body doesn’t make enough insulin or uses insulin poorly. It often begins around middle age and is strongly tied to obesity. People who develop type 2 diabetes may have normal fasting blood glucose concentrations but high postprandial blood sugar values. Over time, they’ll need higher doses of insulin than people without diabetes to keep their blood sugars within acceptable ranges.
The exact mechanism of how diabetes develops remains unclear, but scientists know that it involves both genetic factors and environmental influences like diet, exercise and stress. Scientists believe that a combination of genetics and certain lifestyle habits can set the stage for developing type 2 diabetes. Studies suggest that a common variant of the FTO gene, which encodes a protein involved in fat metabolism, plays a role in predisposing individuals to type 2 diabetes.
Other studies have suggested that low physical activity or excessive weight gain, particularly in adults, might contribute to the development of type 2 diabetes. Some research suggests that inflammation of the lining of the digestive tract may play a role in causing insulin resistance. However, evidence of this link is still inconclusive.
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